Search results for "B-1 cell"

showing 10 items of 18 documents

Inflammatory demyelination induces ependymal modifications concomitant to activation of adult (SVZ) stem cell proliferation

2017

Ependymal cells (E1/E2) and ciliated B1cells confer a unique pinwheel architecture to the ventricular surface of the subventricular zone (SVZ), and their cilia act as sensors to ventricular changes during development and aging. While several studies showed that forebrain demyelination reactivates the SVZ triggering proliferation, ectopic migration, and oligodendrogenesis for myelin repair, the potential role of ciliated cells in this process was not investigated. Using conventional and lateral wall whole mount preparation immunohistochemistry in addition to electron microscopy in a forebrain-targeted model of experimental autoimmune encephalomyelitis (tEAE), we show an early decrease in num…

0301 basic medicineEpendymal CellCell divisionCellSubventricular zoneBiologyCell biologyB-1 cell03 medical and health sciencesCellular and Molecular NeuroscienceMyelin030104 developmental biology0302 clinical medicinemedicine.anatomical_structurenervous systemNeurologymedicineMotile ciliumStem cellNeuroscience030217 neurology & neurosurgeryGlia
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Unique Organization of the Nuclear Envelope in the Post-natal Quiescent Neural Stem Cells

2017

Summary Neural stem cells (B1 astrocytes; NSCs) in the adult ventricular-subventricular-zone (V-SVZ) originate in the embryo. Surprisingly, recent work has shown that B1 cells remain largely quiescent. They are reactivated postnatally to function as primary progenitors for neurons destined for the olfactory bulb and some corpus callosum oligodendrocytes. The cellular and molecular properties of quiescent B1 cells remain unknown. Here we found that a subpopulation of B1 cells has a unique nuclear envelope invagination specialization similar to envelope-limited chromatin sheets (ELCS), reported in certain lymphocytes and some cancer cells. Using molecular markers, [3H]thymidine birth-dating, …

0301 basic medicineNuclear EnvelopeV-SVZBiologyBiochemistry*nuclear ELCSArticleMice03 medical and health sciences*neural stem cellsNeural Stem CellsLateral VentriclesGeneticsAnimalsquiescenceProgenitor celllcsh:QH301-705.5Cells CulturedGeneticslcsh:R5-920*quiescencenuclear envelope invaginationsCell CycleCell Biology*V-SVZnuclear ELCS*nuclear envelope invaginationsEmbryonic stem cellChromatinNeural stem cellOlfactory bulbCell biologyChromatinB-1 cellAdult Stem Cells030104 developmental biologylcsh:Biology (General)nervous systemAstrocytesCancer celllcsh:Medicine (General)Developmental BiologyAdult stem cell
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Adult Neurogenesis Is Sustained by Symmetric Self-Renewal and Differentiation

2018

Somatic stem cells have been identified in multiple adult tissues. Whether self-renewal occurs symmetrically or asymmetrically is key to understanding long-term stem cell maintenance and generation of progeny for cell replacement. In the adult mouse brain, neural stem cells (NSCs) (B1 cells) are retained in the walls of the lateral ventricles (ventricular-subventricular zone [V-SVZ]). The mechanism of B1 cell retention into adulthood for lifelong neurogenesis is unknown. Using multiple clonal labeling techniques, we show that the vast majority of B1 cells divide symmetrically. Whereas 20%-30% symmetrically self-renew and can remain in the niche for several months before generating neurons, …

0301 basic medicineTime FactorsNeurogenesis1.1 Normal biological development and functioningCellventricular-subventricular zoneMice TransgenicCell Counttime-lapse imagingSelf renewalBiologyself-renewalRegenerative MedicineMedical and Health SciencesTransgenicMice03 medical and health sciencesLateral ventricleslineage tracingNeural Stem CellsInterneuronsUnderpinning researchGeneticsmedicineAnimalsHumansCell Self RenewalB1 cellsagingdivision modeNeurogenesisNeurosciencesCell DifferentiationCell BiologyBiological SciencesStem Cell ResearchNeural stem cellCell biologysymmetric divisionB-1 cell030104 developmental biologymedicine.anatomical_structureNeurologicalMolecular MedicineStem Cell Research - Nonembryonic - Non-HumanStem cellDevelopmental BiologyAdult stem cell
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A double-negative (IgD−CD27−) B cell population is increased in the peripheral blood of elderly people

2009

The T cell branch of the immune system has been extensively studied in the elderly and it is known that the elderly have impaired immune function, mainly due to the chronic antigenic load that ultimately causes shrinkage of the T cell repertoire and filling of the immunologic space with memory T cells. In the present paper, we describe the IgD(-)CD27(-) double-negative B cell population which (as we have recently described) is higher in the elderly. Most of these cells were IgG(+). Evaluation of the telomere length and expression of the ABCB1 transporter and anti-apoptotic molecule, Bcl2, shows that they have the markers of memory B cells. We also show that these cells do not act as antigen…

AdultAgingATP Binding Cassette Transporter Subfamily BT cellAntigens CD19B-Lymphocyte Subsetschemical and pharmacologic phenomenaYoung AdultB lymphocyte Immunosenescence IgD CD27 Elderly Immunologic memorymedicineHumansCytotoxic T cellATP Binding Cassette Transporter Subfamily B Member 1IL-2 receptorCD40 AntigensCD154Antigen-presenting cellCells CulturedAgedAged 80 and overSettore MED/04 - Patologia Generalebusiness.industryAge FactorsHLA-DR AntigensImmunoglobulin DMiddle AgedTelomereFlow CytometryAcquired immune systemTumor Necrosis Factor Receptor Superfamily Member 7B-1 cellKi-67 Antigenmedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2ImmunologyB7-1 AntigenbusinessImmunologic MemoryCD80Developmental BiologyMechanisms of Ageing and Development
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Trafficking phenotype and production of granzyme B by double negative B cells (IgG(+)IgD(-)CD27(-)) in the elderly.

2013

The impairment of humoral immune response in elderly humans has been extensively demonstrated. We have reported the increase of memory B cells (IgG(+)IgD(-)CD27(-), double negative, DN) population in the elderly, in which there is also a typical inflammatory micro-environment. In order to evaluate whether this pro-inflammatory status could influence the trafficking phenotype of naïve/memory B cells, we have assessed the expression of CCR7, CCR6, CXCR3, CXCR4, CXCR5 and CD62L on naïve/memory B cell subpopulations in young and elderly subjects. Moreover, the combination of pro-inflammatory interleukin-21 (IL-21) and B cell receptor (BCR) stimulation enables B cells to produce and secrete gran…

AdultAgingChemokine receptorNaive B cellB-cell receptorB-Lymphocyte Subsetschemical and pharmacologic phenomenaBiologyCXCR3BiochemistryGranzymesEndocrinologyImmune systemElderlyIL-21GeneticsHumansSettore MED/05 - Patologia ClinicaL-SelectinMemory B cellMolecular BiologyAgedAged 80 and overReceptors CXCRSettore MED/04 - Patologia GeneraleB lymphocyteGranzyme BInterleukinshemic and immune systemsImmunoglobulin DCell BiologyInflamm-agingTumor Necrosis Factor Receptor Superfamily Member 7B-1 cellGranzyme BImmunosurveillancePhenotypeImmunoglobulin GImmunology
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B cell immunosenescence: different features of naive and memory B cells in elderly.

2011

Elderly people show a reduced protection against new infections and a decreased response to vaccines as a consequence of impairment of both cellular and humoral immunity. In this paper we have studied memory/naive B cells in the elderly, evaluating surface immunoglobulin expression, production of the pro- and anti-inflammatory cytokines, tumor necrosis factor (TNF)-α and interleukin (IL)-10, and presence of somatic hypermutation, focusing on the IgG(+)IgD(-)CD27(-) double negative (DN) B cells that are expanded in the elderly. Our results show that naive B cells from young donors need a sufficiently strong stimulus to be activated "in vitro", while naive B cells from old subjects are able t…

AdultAgingNaive B cellSomatic hypermutationImmunoglobulinsInflammationBiologyLymphocyte ActivationElderlymedicineHumansCytokineB cellCellular SenescenceAgedSettore MED/04 - Patologia GeneraleAged 80 and overB-LymphocytesHypermutationIonomycinGerminal centerImmunosenescenceMiddle AgedMemory B cellsInterleukin-10B-1 cellInterleukin 10medicine.anatomical_structureImmunologyTetradecanoylphorbol AcetateGeriatrics and GerontologyGerontologyCell agingImmunologic MemoryBiogerontology
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A case of agammaglobulinemia characterized by a defect of B-lymphocyte-differentiation to plasma cells

1974

Humoral and cell-mediated immune reactions of a 36-year old patient with a late onset of an agammaglobulinaemia and of his immediate relatives have been investigated. The immunoglobulins gamma-G, gamma-A, gamma-M, gamma-E and gamma-D, and plasma cells in the bone marrow could not be detected in the patient's serum. Immune reactions with PPD and tetanus toxoid as well as the stimulation of the lymphocytes with PHA indicate that the T-cell system was intact. The examination of the peripheral blood lymphocytes by immunofluorescence techniques gave a normal number of B-lymphocytes. These findings could be corroborated by the results of lymphocyte stimulations with anti-IgG. The data suggest tha…

AdultMaleLymphocytePlasma CellsFluorescent Antibody TechniqueImmunoglobulinsStimulationLymphocyte ActivationImmunofluorescenceAgammaglobulinemiaBone MarrowLectinsDrug DiscoveryTetanus ToxoidmedicineHumansHypersensitivity DelayedChildGenetics (clinical)SkinB-Lymphocytesbiologymedicine.diagnostic_testTuberculin TestTetanusToxoidCell DifferentiationImmunoglobulin DGeneral Medicinemedicine.diseaseImmunoglobulin AB-1 cellmedicine.anatomical_structureImmunoglobulin MImmunoglobulin GImmunologybiology.proteinMolecular MedicineBone marrowAntibodyKlinische Wochenschrift
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B cells in the aged: CD27, CD5, and CD40 expression.

2003

Ageing is characterized by numerous changes in lymphocyte subpopulations. In the present paper we have focused on B cells carrying the surface markers CD27, CD5 and CD40. CD27 is considered a marker of primed (memory) cells and its engagement promotes the differentiation of memory B cells into plasma cells. CD5 is expressed on B1 cells, which are considered to be responsible for T cell-independent antibody production other than autoantibodies. The CD40 molecule binds CD40L (CD154) and is necessary for T-dependent antibody responses. Here we show that the absolute number of CD5+ and CD40+ B cells is decreased in the elderly, while CD27+ B lymphocytes only marginally decrease in centenarians.…

Adultmedicine.medical_specialtyAgingNaive B cellchemical and pharmacologic phenomenaCD5 AntigensNatural killer cellInterleukin 21immune system diseasesInternal medicinemedicineHumansLymphocyte CountCD154CD40 AntigensAntigen-presenting cellAgedAged 80 and overB-LymphocytesCD40biologyhemic and immune systemsMiddle AgedMolecular biologyTumor Necrosis Factor Receptor Superfamily Member 7B-1 cellmedicine.anatomical_structureEndocrinologybiology.proteinInterleukin 12BiomarkersDevelopmental BiologyMechanisms of ageing and development
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Direct Cellular Interaction with Activated CD4+T Cells Overcomes Hyporesponsiveness of B-Cell Chronic Lymphocytic Leukemiain Vitro

1998

The proliferative response of clonal B cells from patients with chronic lymphocytic leukemia (B-CLL) is drastically reduced compared to normal B lymphocytes stimulated via the B cell antigen receptor complex or by CD40 ligation. In the present study we demonstrate that hyporesponsiveness of CLL-B cells can be overcome by stimulatory pathways mediated by activated CD4(+) T cells. In contrast to CD40 ligation, costimulation with activated T cells promotes a proliferative response in CLL-B cells identical to that in normal B cells. Furthermore, coculture with activated T cells improved survival of CLL-B cells in vitro. Differentiation of CLL-B cells into IgM producing cells was promoted, as we…

CD4-Positive T-LymphocytesImmunologyB-cell receptorLymphocyte ActivationInterleukin 21Antigens CDhemic and lymphatic diseasesHumansCytotoxic T cellIL-2 receptorCD40 AntigensAntigen-presenting cellCells CulturedB-LymphocytesCD40biologyZAP70Cell DifferentiationLeukemia Lymphocytic Chronic B-CellCell biologyB-1 cellImmunoglobulin MAntigens Surfacebiology.proteinInterleukin-2Cell DivisionCellular Immunology
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Immune Control in Hepatocellular Carcinoma Development and Progression: Role of Stromal Cells

2014

Immune control of hepatocellular carcinoma (HCC) is executed by effector immune cells, which efficiently eliminate malignant transformed cells. However, progression of HCC clearly documents failure of tumor immune control, which led to the concept of immune subversion by the tumor environment. Particularly tumor-associated stromal cells cooperate within an inflammatory network, which is responsible for immune privilege. The stromal cell composition matures during tumor growth and is derived from surrounding noncancerous tissue or from circulating cells recruited to the tumor site. Therefore, immunosuppressive stromal cells represent heterogeneous cell lineages, including myeloid cells, lymp…

Carcinoma HepatocellularStromal cellmedicine.medical_treatmentAdaptive ImmunityBiologyLymphocytes Tumor-InfiltratingImmune systemCancer immunotherapyImmune privilegeTumor MicroenvironmentLymph node stromal cellmedicineAnimalsHumansTumor microenvironmentHepatologyLiver Neoplasmsbiochemical phenomena metabolism and nutritionAcquired immune systemImmunity InnateB-1 cellImmunologyCytokinesbacteriaTumor EscapeImmunotherapyStromal CellsSignal TransductionSeminars in Liver Disease
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